Pain Overview
Pain is an unpleasant sensory and emotional experience associated with actual or potential tissue damage, or described in terms of such damage. Pain is:
- Distinct from nociception
Process of nervous system transmission of a noxious stimuli. - Divided therapeutically and mechanistically into:
- Acute pain
- Chronic pain
Pain lasting beyond the time expected for healing following an injury, occurring due to changes in both peripheral and central nervous systems, as well as psychologically.
- Management is important as pain:
- Causes suffering
- Limits rehabilitation
Adequate analgesia facilitates restoration of normal function. - Leads to secondary complications
- Catecholamine release
- ↑ MVO2
Ischaemia. - Fatigue
- ↑ MVO2
- Stasis
- Atelectasis/pneumonia
- DVT
- Catabolic state
- Progresses to chronic pain
- Catecholamine release
Pain Assessment
Use the PASS assessment:
- Pain history
- Somatic
- Neuropathic
- Analgesia and other drug substances
- Stress and other psychology
- Surgical factors and biological causes
Key components of a pain history include:
- Site
- Location
- Radiation
- Circumstances
Associated trauma, procedures. - Character
Key descriptors. - Intensity
Including:- At rest
- With movement
- Temporal factors
- Duration
- Intensity over time
- Continuous or intermittent
- Aggravating or relieving factors
- Associated symptoms
- Effect of pain on:
- Activities of daily living
- Sleep
- Treatment
- Current and previous medications
- Doses
- Frequency of use
- Efficacy
- Adverse effects
- Other treatment
- Health professionals consulted
- Current and previous medications
- Relevant medical history
- Factors influencing patients symptomatic treatment
- Belief around cause
- Knowledge, expectations, and preferences for management
- Expected outcome
- Reduction in pain required for patient satisfaction
- Coping responses
- Family expectations
Pain Management
Goals of pain management include:
- ↓ Pain initiation
- Inhibit neurotransmission
- Prevent sensitisation
- Central
- Peripheral
- Psychological preparation
- Set expectations
- Control pain
Mechanism-Based Approach
A mechanism-based approach uses a combination of agents to interrupt nociception at multiple levels of the pain fibre. In general, consider:
Key terms:
- Preventative analgesia
Analgesic effect lasts longer than expected duration of drug effet. - Preemptive analgesia
Analgesia provided prior to nociceptive stimulus.
- Allergies and drug intolerances
- Pre-existing pain syndrome or drug exposure
- Nature of painful stimulus
- Expected duration
- Expected magnitude
- Available routes of administration
Then consider an escalating level of:
- Single analgesia
- Paracetamol
- NSAID if appropriate
- Regional techniques (or epidural)
- Non-pharmacological adjuncts
- Requirement for an antineuropathic
- Short-acting opioid
- Oral PRN
- Oxycodone
- Morphine
- Sublingual PRN
- Buprenorphine
- IV PCA
- Oxycodone
- Morphine
- Fentanyl
- Buprenorphine
- Oral PRN
- Pharmacological adjuncts
- Ketamine infusion
- Lignocaine infusion
- Clonidine
- Regular opioid
Situation-Based Approach
A situation-based approach is used when the presentation fits a particular pain syndrome.
Acute Neuropathic Pain
- First line
- Ketamine
- Titrate up with 5mg boluses to effect
- Maintain at 0.1mg/kg/hr IV or SC infusion
- Ketamine
- Second line
- Lignocaine
- Bolus 1-1.5mg/kg
- Maintain at 1-2mg/min
- Lignocaine
Chronic Neuropathic Pain
- First line
- TCAs
- Commence at 5-10mg and ↑ slowly to optimise acceptability
- Effective dose usually 10-100mg
- Slow onset and trial for at least 2 weeks, and ideally 2 months
- Amitryptyline superior if sleep disturbance an issue; give nocte
- Nortriptyline less sedating
- Adverse effects significant
- SSRIs/SNRIs
- Duloxetine 30mg daily, ↑ up to 60mg
- Venlafaxine 37.5mg, ↑ to 75mg, up to 225mg daily
- TCAs
- Second line
α2-delta ligands:- Pregabalin
75mg up to 300mg BD, start at 25-50mg if frail. - Gabapentin
- Pregabalin
- Third line
- Tramadol
- Tapentadol
Phantom Limb Pain
- First line
- Calcitonin
100ug daily SC/ for at least 3 days. Antiemetic prophylaxis.
- Calcitonin
- Second line
Antineuropathics as above.
References
- Schug SA, Palmer GM, Scott DA, Alcock M, Halliwell R, Mott JF; APM:SE Working Group of the Australian and New Zealand College of Anaesthetists and Faculty of Pain Medicine (2020), Acute Pain Management: Scientific Evidence (5th edition), ANZCA & FPM, Melbourne.
- Rigg JR, Jamrozik K, Myles PS, Silbert BS, Peyton PJ, Parsons RW, et al. Epidural anaesthesia and analgesia and outcome of major surgery: a randomised trial. The Lancet. 2002 Apr 13;359(9314):1276–82.
- N. Meylan, N. Elia, C. Lysakowski, M. R. Tramèr. Benefit and risk of intrathecal morphine without local anaesthetic in patients undergoing major surgery: meta-analysis of randomized trials. BJA: British Journal of Anaesthesia, Volume 102, Issue 2, 1 February 2009, Pages 156–167.
- McLeod GA, Cumming C. Thoracic epidural anaesthesia and analgesia. Continuing Education in Anaesthesia Critical Care & Pain, Volume 4, Issue 1, 1 February 2004, Pages 16–19.
- Schug SA, Palmer GM, Scott DA, Halliwell R, Trinca J. Acute pain management: scientific evidence, fourth edition, 2015. Med J Aust. 2016 May 2;204(8):315–7.
- Russell T, Mitchell C, Paech MJ, Pavy T. Efficacy and safety of intraoperative intravenous methadone during general anaesthesia for caesarean delivery: a retrospective case-control study. International Journal of Obstetric Anesthesia. 2013 Jan;22(1):47–51.
- Western Australian Therapeutic Advisory Group. Guidelines for the Pharmacological Treatment of Neuropathic Pain. 2017.