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Blast Trauma

Explosions occur due to a rapid release of chemical energy and cause harm via three mechanisms:

Explosions: Pressure-over-Time

Explosions produce three key effects:

  • Supersonic pressure wave
    Rapid ↑ followed by ↓ of atmospheric pressure without mass particle movement.
    • ↑ In enclosed spaces due to reflection
  • Heat
    Blast wind created by gases produced during the explosion.
  • Gas

Epidemiology and Risk Factors

Pathophysiology

Aetiology

Clinical Features

Primary blast injury:

  • Air filled organs
    • Lung
      • Pneumothorax
      • Lung contusion
      • Alveolar haemorrhage
      • Blast lung
        Alveolar consolidation with fluid and cellular debris.
        • May lead to alveolar-pulmonary venous fistuale and air embolism
    • Tympanic membrane rupture
    • Hollow visceral rupture
  • Globe rupture
  • Mild TBI

Secondary blast injury:

  • Penetrating injury
  • Soft-tissue trauma
  • Traumatic amputation

Tertiary blast injury:

  • Blunt and penetrating injury
  • TBI
  • Crush injury
  • Entrapment

Diagnostic Approach and DDx

Investigations

Bedside:

Laboratory:

Imaging:

Other:

Management

Resuscitation:

  • A
    • Intubation
  • B
    • Lung protective ventilation
      • Minimise Pip
        Risk of systemic air embolism due to direct alveolar-venous communication.

Specific therapy:

  • Pharmacological
  • Procedural
  • Physical

Supportive care:

Disposition:

Preventative:

Marginal and Ineffective Therapies

Anaesthetic Considerations

Complications

  • B
    • ARDS
  • C
    • Air embolism
  • D
    • Permanent hearing loss
    • Tinnitus
    • Abnormal vestibular function

Prognosis

Death:

  • 70% occur within 5 minutes of injury due to catastrophic:
    • Haemorrhage
    • Brain injury

Key Studies


References

  1. Bersten, A. D., & Handy, J. M. (2018). Oh’s Intensive Care Manual. Elsevier Gezondheidszorg.