Salicylates

Salicylates include aspirin and Oil of Wintergreen. The Oil has ↑↑ toxic potential due to ↑ drug concentration.

Salicylate overdose classically causes:

Nausea and vomiting occur due to irritation of both gut mucosa and the medullary chemoreceptor trigger zone.

Epidemiology and Risk Factors

Pathophysiology

Aetiology

Clinical Features

Initial:

  • Tinnitus
  • Nausea, vomiting
  • Abdominal pain

Severe:

  • Pulmonary oedema
  • ARDS
  • Seizures
  • Coma
  • Acidaemia
  • Hyper or hypoglycaemia
  • Hypokalaemia

Assessment

History:

  • Dose
    Risk assessment:
    • <150mg/kg: Minimal
    • 150-300mg/kg: Mild-moderate
      Recommend activated charcoal.
    • >300mg/kg: Severe intoxication, metabolic acidosis and neurological features expected
    • >500mg/kg: Potentially lethal
      Consider gastric lavage.

Exam:

Investigations

Many bedside ABG machines mistake salicylate for chloride and incorrectly report a hyperchloraemic NAGMA.

Bedside:

  • ABG
    • Respiratory alkalosis
    • HAGMA

Laboratory:

  • Blood
    • Salicylate level
      Correlate poorly with toxicity but can be used to identify trend of absorption.
    • UEC
      • Monitor for hypokalaemia
    • Paracetamol level
    • BSL

Imaging:

Other:

Diagnostic Approach and DDx

Management

  • Activated charcoal
  • Treat seizures
  • Urinary alkalinisation or RRT

Resuscitation:

  • Pulmonary oedema
    • CPAP
      NIV or intubation, depending on conscious state.
  • C
    • Fluid resuscitation
      GIT and insensible losses.
  • D
    • Seizures
      • Benzodiazepines

Specific therapy:

Activated charcoal and urinary alkalinisation are covered in detail under Poisoning.

  • Pharmacological
    • Activated charcoal
      Up to 8 hours following overdose.
    • Alkalinisation
      • ↑ Urine pH traps salicylate ions in urine, ↑ elimination
      • ↑ Blood pH prevents salicylate movement into CSF
  • Procedural
    • RRT
      • ↑ Clearance
      • Allows control of volume when bicarbonate is ↑
  • Physical

Supportive care:

  • B
    • Hyperventilation
      To aid alkalinisation.
  • F
    • Correct hypokalaemia
      Hypokalaemia will ↑ DCT K+/H+ exchange, acidifying urine.

Disposition:

Preventative:

Marginal and Ineffective Therapies

Anaesthetic Considerations

Complications

Prognosis

Key Studies


References

  1. Bersten, A. D., & Handy, J. M. (2018). Oh’s Intensive Care Manual. Elsevier Gezondheidszorg.