Jet Ventilation

Jet ventilation is the use of high-pressure (1-3 bar), high frequency ventilation to facilitate oxygenation and ventilation. Jet ventilation:

Mechanisms

May be either:

  • Manual
    Low frequency.
  • Automated
    Low (8 bpm) to high (up to 300bpm) frequency.
    • Reduced barotrauma compared to manual jet ventilation

Manual Jet Ventilation

Initial setup:

  • Driving pressure: 3.5 bar (0-3.5 bar)
  • Respiratory rate: ~8
  • Inspiratory time: ~1s, aiming 4-5s expiration
    Keep hand or eyes on chest throughout to ensure full expiration occurs - high risk for barotrauma.

Automated Jet Ventilators

  • Deliver heated, humidified gas jets in a square wave at 1-10Hz
  • Clinician can control driving pressure, frequency, inspiratory time, and composition of gas
  • Have alarms and emergency shutdown devices in presence of high airway pressures

Advantages:

  • Minimises vocal cord, bronchial, and mediastinal excursion
  • Can be passed through surgical field to jet into trachea
  • Less haemodynamic impact compared with IPPV
  • CO can be augmented using ECG synchronisation
  • Avoids potential for ETT fire

Initial Setup:

  • Driving pressure: 2 bar
    1-3 bar acceptable range.
  • Frequency: 200/min
    150-300cpm. ↑ frequency reduces tidal volume.
  • FiO2: 1.0
  • I-time: 20%
    Adjust 20-70%. ↑ inspiratory time ↑ tidal volume.
  • Pause pressure: 2mbar
    10-40mbar. This is the pressure measured at the tip of the ventilation catheter ~1ms before the next jet.

Adjustment:

  • ↑ oxygenation and ventilation requires ↑ pressure, not rate

Intraoperative Management:

  • Check for movements of the chest and abdomen
  • Check CO2 if sampling port connected
    Not an accurate assessment of arterial CO2, and is easily lost if the port is blocked or kinked.
  • Turn off HFJV prior to removing catheter to prevent contamination

Techniques

Supraglottic ventilation usually required for posterior cord lesions.

May be:

Supraglottic{#supra}

Jet kept in glottis and gas jetted through the glottis. * Pros: * Cheap * No tube in surgical field * Cons: * Greater rate of ↑ in airway pressure compared to subglottic ventilation
Counter-intuitive, but occurs due to: * Venturi effect
Greater entrained air with supraglottic approach ↑ tidal volume and reduces FiO2. * Double jetting
Supraglottic jet is reflected from glottis, ↑ supraglottic pressure and impeding expiratory flow. * Expiratory impedance
Expiration can only occur during the expiratory phase of the cycle, which is a function of the I:E ratio of the jet ventilator. * No CO2 monitoring * Cannot monitor peak airway pressure * Inadequate ventilation
If aim is incorrect. Monitor: * Sound * Chest movement * Movement of surgical field ↑ compared to subglottic approaches Can’t ventilate whilst lasering.

Infraglottic

Jet catheter placed in trachea via glottis:

  • Pros
    • Slow rate of pressure ↑
    • Consistent FiO2
      Minimal entrainment.
    • Expiration can occur through a subglottic stenosis throughout the respiratory cycle, reducing expiratory impedance
  • Cons
    • Requires an intra-tracheal catheter
      Obstructs surgical field.
      • Hunsaker Monjet
        • Basket assists central direction of jet
        • Laser-resistant
        • Should be placed >7.5cm post the cords so the monitoring port is at an adequate depth
        • Monitoring port easily blocked or kinked
      • LaserJet
        • Similar to Hunsaker but without the basket
        • Monitoring lumen for airway pressure and CO2

Transtracheal

Jet catheter placed in trachea via front of neck:

  • Pros
    • Anaesthetic advantages of infraglottic jet ventilation
    • Surgical advantages of supraglottic jet ventilation
  • Cons
    • Requires front of neck access
    • Risk of gas trapping with small diameter stenoses
      Air pressure exceeds that of infraglottic jetting when stenosis diameter is <4.5mm.

Complications

  • Barotrauma
    May lead to subcutaneous emphysema, pneumothorax, or pneumomediastinum. Occurs due to:

    • Excessive inspiratory pressures
    • Prolonged inspiratory time
    • Outflow obstruction
  • Gas trapping
    Outflow obstruction leading to rise in airway pressures and occlusion of venous return.

  • Dry gas trauma
    Occurs if jetting without humidified gas. Leads to epithelial necrosis, mucous buildup, and airway pluggin.

  • Hyperoxia
    Due to fall in FiO2 or ↓ airway pressure and atelectasis.

  • Hypercapnoea
    Can be avoided by ↑ driving pressure (to ↑ tidal volume) and ↓ frequency (to ↑ E time).

    • Malignancy
      Theoretical risk of spreading tumour cells.
    • Risk of airborne contamination if papilloma; wear masks